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A form of frontotemporal epilepsy in which the lesion develops in the opercular zone of the brain.

The clinical picture of convulsive seizures is represented by profuse salivation, clonic convulsions in the muscles of arcoxia pills, neck and larynx, a tingling sensation in the throat and half of the face. The most relevant is the division of temporal lobe epilepsy into.

The most common and frequently encountered form of symptomatic disease is medial, or mesial, temporal sclerosis.

Also in medicine there is such a thing as structural temporal lobe epilepsy, which means the morphological characteristics of etoricoxib condition. Idiopathic. With this form, there are no structural changes in the brain tissues. It develops under the influence of genetic factors. Cryptogenic. One of the most dangerous forms of temporal lobe epilepsy.

The disease tends to progress.

She is characterized by complex attacks, accompanied by pain in the heart and abdomen. People with cryptogenic temporal lobe epilepsy may become panicky and have olfactory and visual disturbances. Because of the feeling of fear, patients may take unreasonable actions. The occurrence of temporal lobe epilepsy can be triggered by a number of etoricoxib factors. They can be divided into two groups.

These include those that affect the fetus at the stage of its intrauterine development.

They also include generic influencing factors. The impetus for the development of temporal lobe epilepsy can be an injury received during childbirth, oxygen starvation of the fetus, various intrauterine infections such as measles, rubella, syphilis and others, as well as asphyxia of the baby at the time of birth.

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Underthese developmental mechanisms imply the occurrence of epileptic activity due to severe traumatic brain injury and infections affecting the central nervous system. This group of factors also includes hemorrhagic and ischemic stroke. Also, brain lesions such as large tumors, hematoma, cerebral aneurysm, sclerosis and abscess contribute to the progression of the disease. In more than half of the cases, the disease develops due to medial temporal sclerosis. This ailment can both cause epilepsy and be the result of prolonged convulsive seizures.

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Cryptogenic epilepsy is one of the most complex chronic pathologies of the central nervous system, the development of which is due to unidentified or uncertain causes. The disease is manifested by regularly recurring specific seizures. The cryptogenic form is found in almost 50% of patients suffering from epileptic seizures. Depending on the pathological focus, epilepsy can occur in such proportions as.

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The frontal form is often accompanied by sudden attacks lasting in short series.

In rare situations, secondary generalization occurs due to damage to brain cells.

Pathology is always accompanied by increased activity in the region of the crown, which is responsible for sensitive functions. Affected people experience unusual sensory sensations, such as. Accompanied by partial seizures, refers to the rarest form.

However, scientists concluded that the development of the disease can be triggered by the following factors.

The most likely cause of the pathology is considered to be a genetic predisposition. The clinical picture of cryptogenic epilepsy is manifested by the following symptoms.

The leading symptom complex of cryptogenic focal epilepsy is recurrent partial paroxysm.

The nature of simple partial seizures is motor, sensory, vegetative, somatosensory, accompanied by auditory, visual, olfactory or gustatory hallucinations and mental disorders. Myoclonic epilepsy is a type of epileptic seizures. It has a softer flow. The disease first appears in infants or young children. The debut of myoclonic epilepsy in adulthood is atypical. Causes of myoclonic epilepsy.

Each variety of cryptogenic epilepsy has its own characteristic features and symptomatic manifestations that allow differentiating one form from another.

Currently, the exact causes of myoclonic epilepsy have not been established. However, doctors identify several predisposing factors. Among them. hereditary predisposition. Certain types of myoclonic epilepsy are hereditary. For example, Unferricht-Lundborg disease, Drave syndrome.

If one of the relatives was diagnosed with epilepsy, then subsequent generations are at risk.

The probability of occurrence of the disease is 20-30%. intrauterine infection. Some infectious agents are able to cross the placental barrier. As a result, the risk of developing not only myoclonic epilepsy, but also mental disorders and developmental defects increases. Myoclonic epilepsy develops at the end of the 2nd or 3rd trimester.

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